---
scientific_name: Glycine max
gene_symbols:
  - GmChlI1a
gene_symbol_long: Mg-Chelatase Subunit Chl1a
gene_model_pub_name: Glyma.13G232500
gene_model_full_id: glyma.Wm82.gnm2.ann1.Glyma.13G232500
confidence: 4
curators:
  - Marlene Dorneich-Hayes
  - Scott Kalberer
comments:
  - The MinnGold and T219H phenotypes cosegregate with ChlI1a. Transformation of WT ChlI1a into mutant soy reinstated normal phenotype.
phenotype_synopsis: Defective Mg-Chetelase is responsible for chlorophyll deficient MinnGold (y11-2) and T219H (y11) phenotypes.
traits:
  - entity_name: magnesium chelatase activity
    entity: GO:0016851
  - entity_name: leaf chlorophyll content
    entity: TO:0012002
references:
  - citation: Campbell, Mani et al., 2014
    doi: 10.1534/g3.114.015255
    pmid: 25452420
---
scientific_name: Glycine max
gene_symbols:
  - GmChlI1b
gene_symbol_long: Mg-Chelatase Subunit Chl1b
gene_model_pub_name: Glyma.15G080200
gene_model_full_id: glyma.Wm82.gnm2.ann1.Glyma.15G080200
confidence: 4
curators:
  - Marlene Dorneich-Hayes
  - Scott Kalberer
comments:
  - The CD-5 phenotype cosegregates with ChlI1b.
phenotype_synopsis: Defective Mg-chetelase is responsible for chlorophyll deficient CD-5 phenotype.
traits:
  - entity_name: magnesium chelatase activity
    entity: GO:0016851
  - entity_name: leaf chlorophyll content
    entity: TO:0012002
references:
  - citation: Campbell, Mani et al., 2014
    doi: 10.1534/g3.114.015255
    pmid: 25452420